An attempt towards the rectification of the rot that has set in the social and national life of the country is being made, with great enthusiasm, through judicial activism. However judicial activism alone does not suffice for the rectification of this all-pervading malaise. What is urgently needed is the support of other social agencies/institutions. A very important role in this matter can be played by media activism, the topic under discussion today.
It is undoubtedly true that the position of media or journalism is not that of a mission but of a commercial industry. The truth of the matter, however, is that our present journalism is used to presenting only half side of the picture. This is the root cause of all our problems. In view of the present circumstances media activism would amount to present a balanced reporting of the situation abandoning the present policy of selective reporting.
The principle of modern journalism can be understood from this saying: "When a dog bites a man it is no news, but when a man bites a dog, it is news". One practical example of this method is provided by our present journalism which is constantly engaged in giving maximum coverage to any hot news created by an unruly section of Muslims. If the percentage of hot news forms only one percent the percentage of soft news is not less than 99 percent. But the readers of the newspapers aretotally in the dark about this 99 percent of the picture, whereas the one percent is being repeated. Similarly if an extremist Hindu creates hot news, this will find a place in all the newspapers the next day. Whereas even in the Hindu world there is 99 percent soft news while hot news forms not more than one percent.
As a result of this one sided study, unreal opinion is formed by both the communities regarding one another. Taking extreme forms this unreal opinion at times turns into communal riots. The selective reporting of this nature remains a permanent obstacle to the path of national integration.
For the rectification of this state of affairs a powerful journalists organization-as we already have formed for our rights-based on the principles of social responsibilities is required. Media Relations Forum is an organization which aims at working for this goal.
Along with this I should like to put forward a proposal for bringing about an atmosphere of support and cooperation between the newspapers and social workers. Whenever a rumor spreads or a group indulges in any activity which may lead to disrupting peace, social workers should immediately engage themselves in a thorough investigation of the matter and then through the full support of the newspapers the actual version is published in the newspapers. This is the only way to maintain peace and harmony in communities.

Which one of the following is the characteristic of the current reporting（）

A:Commercial reporting. B:Balanced reporting. C:Selective reporting. D:Problem reporting.

Clever genetic detective work may have found out the reason why a near-starvation diet prolongs the life of many animals.
Ronald Kahn at Harvard Medical School in Boston, US, and his colleagues have been able to extend the lifespan (寿命) of mice by 18 percent by blocking the rodent’s (啮齿动物) increase of fat in specific cells. This suggests that thinness-and not necessarily diet-promotes long life in "calorie (热量单位，卡) restricted" animals.
"It’s very cool work," says aging researcher Cynthia Kenyon of the University of California, San Francisco. "These mice eat all they want, lose weight and live longer. It’s like heaven. "
Calorie restriction dramatically extends the lifespan of organisms as different as worms and rodents. Whether this works in humans is still unknown, partly because few people are willing to submit to such a strict diet.
But many researchers hope they will be able to trigger the same effect with a drug once they understand how less food leads to a longer life. One theory is that eating less reduces the increase of harmful things that can damage cells. But Kahn’s team wondered whether the animals simply benefit by becoming thin.
To find out, they used biology tricks to disrupt the insulin (胰岛素) receptor (受体) gene in lab mice—but only in their fat cells. "Since insulin is needed to help fat cells store fat, these animals were protected against becoming fat," explains Kahn.
This slight genetic change in a single tissue had dramatic effects. By three months of age, Kahn’s modified mice had up to 70 percent less body fat than normal control mice, despite the fact that they ate 55 percent more food per gram of body weight.
In addition, their lifespan increased. The average control mouse lived 753 days, while the thin rodents averaged a lifespan of 887 days. After three years, all the control mice had died, but one-quarter of the modified rodents were still alive.
"That they get these effects by just manipulating the fat cells is controversial, "says Leonard Guarente of the Massachusetts Institute of Technology, who studies calorie restriction and aging.
But Guarente says Kahn has yet to prove that the same effect is responsible for increased lifespan in calorie restricted animals. "It might be the same effect or there might be two routes to long life," he points out, "and that would be very interesting./

Ronald Kahn and his colleagues can make mice live longer by （　）

A:offering them less food B:giving them a balanced diet C:disrupting the specific genes in their fat cells D:preventing them growing larger

Clever genetic detective work may have found out the reason why a near-starvation diet prolongs the life of many animals.
Ronald Kahn at Harvard Medical School in Boston, U.S., and his colleagues have been able to extend the lifespan (寿命) of mice by 18 per cent by blocking the rodent’s (啮齿动物) increase of fat in specific cells. This suggests that thinness--and not necessarily diet--promotes long life in "calorie (热量卡) restricted" animals.
"It’s very cool work," says aging researcher Cynthia Kenyon of the University of California, San Francisco. "These mice eat all they want, lose weight and live longer. It’s like heaven."
Calorie restriction dramatically extends the lifespan of organisms as different as worms and rodents. Whether this works in humans is still unknown partly because few people are willing to submit to such a strict diet.
But many researchers hope they will be able to trigger the same effect with a drug once they understand how less food leads to a longer life. One theory is that eating less reduces the increase of harmful things that can damage cells. But Kahn’s team wondered whether the animals simply benefit by becoming thin.
To find out, they used biology tricks to disrupt the insulin (胰岛素) receptor (受体) gene in lab mice, but only in their fat cells. "Since insulin is needed to help fat cells store fat, these animals were protected against becoming fat," explains Kahn.
This slight genetic change in a single tissue had dramatic effects. By three months of age, Kahn’s modified mice had up to 70 per cent less body fat than normal control mice, despite the fact that they ate 55 per cent more food per gram of body weight.
In addition, their lifespan increased. The average control mouse lived 753 days, while the thin rodents averaged a lifespan of 887 days. After three years, all the control mice had died, but one-quarter of the modified rodents were still alive.
"That they get these effects by just manipulating the fat cells is controversial," says Leonard Guarente of the Massachusetts Institute of Technology, who studies calorie restriction and aging.
But Guarente says Kahn has yet to prove that the same effect is responsible for increased lifespan in calorie-restricted animals. "It might be the same effect or there might be two routes to long life," he points out, "and that would be very interesting./
Ronald Kahn and his colleagues can make mice live longer by ______.

A:offering them less food B:giving them a balanced diet C:disrupting the specific genes in their fat cells D:preventing them growing larger

Clever genetic detective work may have found out the reason why a near-starvation diet prolongs the life of many animals.
Ronald Kahn at Harvard Medical School in Boston, US, and his colleagues have been able to extend the lifespan (寿命) of mice by 18 percent by blocking the rodent’s (啮齿动物) increase of fat in specific cells. This suggests that thinness—and not necessarily diet— promotes long life in "calorie (热量单位, 卡) restricted" animals.
"It’s very cool work, "says aging researcher Cynthia Kenyon of the University of California, San Francisco. "These mice eat all they want, lose weight and live longer. It’s like heaven."
Calorie restriction dramatically extends the lifespan of organisms as different as worms and rodents. Whether this works in humans is still unknown, partly because few people are willing to submit to such a strict diet.
But many researchers hope they will be able to trigger the same effect with a drug once they understand how less food leads to a longer life. One theory is that eating less reduces the increase of harmful things that can damage cells. But Kahn’s team wondered whether the animals simply benefit by becoming thin.
To find out, they used biology tricks to disrupt the insulin (胰岛素) receptor (受体) gene in lab mice—but only in their fat cells. "Since insulin is needed to help fat ceils store fat, these animals were protected against becoming fat," explains Kahn.
This slight genetic change in a single tissue had dramatic effects. By three months of age, Kahn’s modified mice had up to 70 percent less body fat than normal control mice, despite the fact that they ate 55 percent more food per gram of body weight.
In addition, their lifespan increased. The average control mouse lived 753 days, while the thin rodents averaged a lifespan of 887 days. After three years, all the control mice had died, but one-quarter of the modified rodents were still alive.
"That they get these effects by just manipulating the fat cells is controversial," says Leonard Guarente of the Massachusetts Institute of Technology, who studies calorie restriction and aging.
But Guarente says Kahn has yet to prove that the same effect is responsible for increased lifespan in calorie-restricted animals. "It might be the same effect or there might be two routes to long life," he points out, "and that would be very interesting./

Ronald Kahn and his colleagues can make mice live longer by （　）.

A:offering them less food B:giving them a balanced diet C:disrupting the specific genes in their fat cells D:preventing them growing larger

Eat More, Weigh Less, Live Longer
Clever genetic detective work may have found out the reason why a near-starvation diet prolongs the life of many animals.
Ronald Kahn at Harvard Medical School in Boston, US, and his colleagues have been able to extend the lifespan (寿命) of mice by 18 per cent by blocking the rodent’s (啮齿动物) increase of fat in specific cells. This suggests that thinness—and not necessarily diet—promotes long life in "calorie (热量卡) restricted" animals.
"It’s very coal work," says aging researcher Cynthia Kenyon of the University of California, San Francisco. "These mice eat all they want, lose weight and live longer. It’s like heaven."
Calorie restriction .dramatically extends the lifespan of organisms as different as worms and rodents. Whether this works in humans is still unknown, partly because few people are willing to submit to such a strict diet.
But many researchers hope they will be able to trigger the same effect with a drug once they understand how less .food leads to a longer life. One theory is that eating less reduces the increase of harmful things that can damage cells. But kahn’s team wondered whether the animals simply benefit by becoming thin:
To find out, they used biology tricks to disrupt the insulin (胰岛素) receptor (受体) gene in lab mice—but only in their fat cells. "Since insulin is needed to help fat cells store fat, these animals were protected against becoming fat," explains Kahn.
This slight genetic change in a single tissue had dramatic effects. By three months of age, Kahn’s modified mice had up to 70 per cent less body fat than normal control mice, despite the fact that they ate 55 per cent more food per gram of body weight.
In addition, their lifespan increased. The average control mouse lived 753 days, while the thin rodents averaged a lifespan of 887 days. After three years, all the control mice had died, but one-quarter of the modified rodents were still alive.
"That they get these effects by just manipulating the fat cells is controversial," says Leonard Guarente of the Massachusetts Institute of Technology, who studies calorie restriction and aging..
But Guarente says Kahn has yet to prove that the same effect is responsible for increased lifespan in calorie-restricted animals. "It might be the same effect or there might be two routes to long life," he points out, "and that would be very interesting./

Ronald Kahn and his colleagues can make mice live longer by（　）.

A:offering them less food. B:giving them a balanced diet. C:disrupting the specific genes in their fat cells. D:preventing them growing larger.

Eat More, Weigh Less, Live Longer
Clever genetic detective work may have found out the reason why a near-starvation diet prolongs the life of many animals.
Ronald Kahn at Harvard Medical School in Boston, US, and his colleagues have been able to extend the lifespan (寿命) of mice by 18 per cent by blocking the rodent’s (啮齿动物) increase of fat in specific cells. This suggests that thinness—and not necessarily diet—promotes long life in "calorie (热量卡) restricted" animals.
"It’s very coal work," says aging researcher Cynthia Kenyon of the University of California, San Francisco. "These mice eat all they want, lose weight and live longer. It’s like heaven."
Calorie restriction .dramatically extends the lifespan of organisms as different as worms and rodents. Whether this works in humans is still unknown, partly because few people are willing to submit to such a strict diet.
But many researchers hope they will be able to trigger the same effect with a drug once they understand how less .food leads to a longer life. One theory is that eating less reduces the increase of harmful things that can damage cells. But kahn’s team wondered whether the animals simply benefit by becoming thin:
To find out, they used biology tricks to disrupt the insulin (胰岛素) receptor (受体) gene in lab mice—but only in their fat cells. "Since insulin is needed to help fat cells store fat, these animals were protected against becoming fat," explains Kahn.
This slight genetic change in a single tissue had dramatic effects. By three months of age, Kahn’s modified mice had up to 70 per cent less body fat than normal control mice, despite the fact that they ate 55 per cent more food per gram of body weight.
In addition, their lifespan increased. The average control mouse lived 753 days, while the thin rodents averaged a lifespan of 887 days. After three years, all the control mice had died, but one-quarter of the modified rodents were still alive.
"That they get these effects by just manipulating the fat cells is controversial," says Leonard Guarente of the Massachusetts Institute of Technology, who studies calorie restriction and aging..
But Guarente says Kahn has yet to prove that the same effect is responsible for increased lifespan in calorie-restricted animals. "It might be the same effect or there might be two routes to long life," he points out, "and that would be very interesting./

Ronald Kahn and his colleagues can make mice live longer by（）。

A:offering them less food. B:giving them a balanced diet. C:disrupting the specific genes in their fat cells. D:preventing them growing larger.

Clever genetic detective work may have found out the reason why a near starvation diet prolongs the life of many animals.
Ronald Kahn at Harvard Medical School in Boston, U.S., and his colleagues have been able to extend the lifespan of mice by 18 per cent by blocking the rodent’s increase of fat in specific cells. This suggests that thinness and hot necessarily diet promotes long life in "calorie restricted" animals.
"It’s very cool work." says aging researcher Cynthia Kenyon of the University of California, San Francisco. "These mice eat all they want, lose weight and live longer. It’s like heaven."
Calorie restriction dramatically extends the lifespan of organisms as different as worms and rodents. Whether this works in humans is still unknown, partly because few people are willing to submit to such a strict diet.
But many researchers hope they will be able to trigger the same effect with a drug once they understand how less food leads to a longer life. One theory is that eating less reduces the increase of harmful things that can damage cells. But Kahn’s team wondered whether the animals simply benefit by becoming thin.
To find out they used biology tricks to disrupt the insulin (胰岛素) receptor (受体) gene in lab mice but only in their fat cells. "Since insulin is needed to help fat cells store fat, these animals were protected against becoming fat," explains Kahn.
This slight genetic change in a single tissue had dramatic effects. By three months of age, Kahn’s modified mice had up to 70 percent less body fat than normal control mice, despite the fact that they ate 55 percent more food per gram of body weight.
In addition, their lifespan increased. The average control mouse lived 753 days, while the thin rodents averaged a lifespan of 887 days. After three years, all the control mice had died, but one-quarter of the modified rodents were still alive.
"That they get these effects by just manipulating the fat cells is controversial," says Leonard Guarente of the Massachusetts Institute of Technology, who studies calorie restriction and aging.
But Guarente says Kahn has yet to prove that the same effect is responsible for increased lifespan in calorie restricted animals. "It might be the same effect or there might be two routes to long life," he points out, "and that would be very interesting./
Ronald Kahn and his colleagues can make mice live longer by ______.

A:offering them less food. B:giving them a balanced diet. C:disrupting the specific genes in their fat cells. D:preventing them growing larger.

Ronald Kahn and his colleagues can make mice live longer by

A:offering them less food. B:giving them a balanced diet. C:disrupting the specific genes in their fat cells. D:preventing them growing larger.

{{B}}第三篇{{/B}}

Ronald Kahn and his colleagues can make mice live longer by

A:offering them less food. B:giving them a balanced diet. C:disrupting the specific genes in their fat cells. D:preventing them growing larger.