Text 2
For almost ten years, Noel Heath and Glenroy Matthew, better known as "Zambo" and "Bobo", have escaped attempts by the United States to extradite them from their homes on the pretty little island of St Kitts to face charges of cocaine trafficking. Their creative legal team has twice taken the case to the Privy Council in London, still the final appeal court for most of Britain’s former Caribbean colonies. Both times, most recently last November, a panel of British law lords ruled that they should be extradited "with the utmost expedition".
"Zambo" and "Bobo" are well-connected in St Kitts. They have lived on bail for a decade, be fore being locked up last month. Their lawyers hit back with a habeas corpus writ, to be heard on January 18th. If that fails, the way is open for officials to put the two on a plane.
For reasons of principle, or of friendships in tight-knit communities, or both, Caribbean countries have been reluctant to extradite their own nationals. The Caribbean has also become something of a heaven for foreigners wanted elsewhere in the world. This may now change. The next important test comes in May, when the Privy Council will rule on Samuel "Ninety" Knowles, a Bahamian who has held out since 2000 against a charge by a grand jury in Florida.
Procedural complexities and powerful lawyers may still stop extraditions. In September in Belize, Dean Barrow, a lawyer who is also the leader of the parliamentary opposition, hedged an American attempt to extradite a drug suspect. He found mistakes in supporting paperwork, which excluded the use of vital wiretap evidence.
Extradition of foreigners, especially to their home country, is often easier. Viktor Kozeny, a Czech-born resident of the Bahamas, has been held in Nassau since October. He is wanted in New York for corruption stemming from the privatisation of Azerbaijan’s oil company, and faces other charges in Prague.
Mr. Kozeny will fight hard. His lawyers include Philip Davis, a member of parliament for the governing party and former legal partner of the prime minister. Even so, the authorities seem reluctant to grant bail. Perhaps that is because Mr. Kozeny holds a pilot’s licence and Irish and Venezuelan passports. He was once a diplomat for Grenada.
Non-citizens are sometimes simply expelled. Two Belizean women picked up $ 50,000 each on the Oprah Winfrey Show in October, their reward for spotting an alleged rapist from the United States who was sent home two days later for trial. It is rarely so quick or easy.

The Caribbean became something of a heaven because（）

A:it has heaven-like scenery. B:it welcomes all people from other parts of the world. C:it protects people from being extradited. D:it is a paradise for criminals.

Clever genetic detective work may have found out the reason why a near-starvation diet prolongs the life of many animals.
Ronald Kahn at Harvard Medical School in Boston, US, and his colleagues have been able to extend the lifespan (寿命) of mice by 18 percent by blocking the rodent’s (啮齿动物) increase of fat in specific cells. This suggests that thinness-and not necessarily diet-promotes long life in "calorie (热量单位，卡) restricted" animals.
"It’s very cool work," says aging researcher Cynthia Kenyon of the University of California, San Francisco. "These mice eat all they want, lose weight and live longer. It’s like heaven. "
Calorie restriction dramatically extends the lifespan of organisms as different as worms and rodents. Whether this works in humans is still unknown, partly because few people are willing to submit to such a strict diet.
But many researchers hope they will be able to trigger the same effect with a drug once they understand how less food leads to a longer life. One theory is that eating less reduces the increase of harmful things that can damage cells. But Kahn’s team wondered whether the animals simply benefit by becoming thin.
To find out, they used biology tricks to disrupt the insulin (胰岛素) receptor (受体) gene in lab mice—but only in their fat cells. "Since insulin is needed to help fat cells store fat, these animals were protected against becoming fat," explains Kahn.
This slight genetic change in a single tissue had dramatic effects. By three months of age, Kahn’s modified mice had up to 70 percent less body fat than normal control mice, despite the fact that they ate 55 percent more food per gram of body weight.
In addition, their lifespan increased. The average control mouse lived 753 days, while the thin rodents averaged a lifespan of 887 days. After three years, all the control mice had died, but one-quarter of the modified rodents were still alive.
"That they get these effects by just manipulating the fat cells is controversial, "says Leonard Guarente of the Massachusetts Institute of Technology, who studies calorie restriction and aging.
But Guarente says Kahn has yet to prove that the same effect is responsible for increased lifespan in calorie restricted animals. "It might be the same effect or there might be two routes to long life," he points out, "and that would be very interesting./

What does the last sentence in the third paragraph imply（　）

A:People like to lose weight, but they do not like to eat less. B:People want to go to heaven, but they do not want to die. C:Mice will go to heaven if they lose weight. D:Mice enjoy losing weight.

Clever genetic detective work may have found out the reason why a near-starvation diet prolongs the life of many animals.
Ronald Kahn at Harvard Medical School in Boston, U.S., and his colleagues have been able to extend the lifespan (寿命) of mice by 18 per cent by blocking the rodent’s (啮齿动物) increase of fat in specific cells. This suggests that thinness--and not necessarily diet--promotes long life in "calorie (热量卡) restricted" animals.
"It’s very cool work," says aging researcher Cynthia Kenyon of the University of California, San Francisco. "These mice eat all they want, lose weight and live longer. It’s like heaven."
Calorie restriction dramatically extends the lifespan of organisms as different as worms and rodents. Whether this works in humans is still unknown partly because few people are willing to submit to such a strict diet.
But many researchers hope they will be able to trigger the same effect with a drug once they understand how less food leads to a longer life. One theory is that eating less reduces the increase of harmful things that can damage cells. But Kahn’s team wondered whether the animals simply benefit by becoming thin.
To find out, they used biology tricks to disrupt the insulin (胰岛素) receptor (受体) gene in lab mice, but only in their fat cells. "Since insulin is needed to help fat cells store fat, these animals were protected against becoming fat," explains Kahn.
This slight genetic change in a single tissue had dramatic effects. By three months of age, Kahn’s modified mice had up to 70 per cent less body fat than normal control mice, despite the fact that they ate 55 per cent more food per gram of body weight.
In addition, their lifespan increased. The average control mouse lived 753 days, while the thin rodents averaged a lifespan of 887 days. After three years, all the control mice had died, but one-quarter of the modified rodents were still alive.
"That they get these effects by just manipulating the fat cells is controversial," says Leonard Guarente of the Massachusetts Institute of Technology, who studies calorie restriction and aging.
But Guarente says Kahn has yet to prove that the same effect is responsible for increased lifespan in calorie-restricted animals. "It might be the same effect or there might be two routes to long life," he points out, "and that would be very interesting./
What does the last sentence in the third paragraph imply

A:People like to lose weight, but they do not like to eat less B:People want to go to heaven, but they do not want to die C:Mice will go to heaven if they lose weight D:Mice enjoy losing weight

Clever genetic detective work may have found out the reason why a near-starvation diet prolongs the life of many animals.
Ronald Kahn at Harvard Medical School in Boston, US, and his colleagues have been able to extend the lifespan (寿命) of mice by 18 percent by blocking the rodent’s (啮齿动物) increase of fat in specific cells. This suggests that thinness—and not necessarily diet— promotes long life in "calorie (热量单位, 卡) restricted" animals.
"It’s very cool work, "says aging researcher Cynthia Kenyon of the University of California, San Francisco. "These mice eat all they want, lose weight and live longer. It’s like heaven."
Calorie restriction dramatically extends the lifespan of organisms as different as worms and rodents. Whether this works in humans is still unknown, partly because few people are willing to submit to such a strict diet.
But many researchers hope they will be able to trigger the same effect with a drug once they understand how less food leads to a longer life. One theory is that eating less reduces the increase of harmful things that can damage cells. But Kahn’s team wondered whether the animals simply benefit by becoming thin.
To find out, they used biology tricks to disrupt the insulin (胰岛素) receptor (受体) gene in lab mice—but only in their fat cells. "Since insulin is needed to help fat ceils store fat, these animals were protected against becoming fat," explains Kahn.
This slight genetic change in a single tissue had dramatic effects. By three months of age, Kahn’s modified mice had up to 70 percent less body fat than normal control mice, despite the fact that they ate 55 percent more food per gram of body weight.
In addition, their lifespan increased. The average control mouse lived 753 days, while the thin rodents averaged a lifespan of 887 days. After three years, all the control mice had died, but one-quarter of the modified rodents were still alive.
"That they get these effects by just manipulating the fat cells is controversial," says Leonard Guarente of the Massachusetts Institute of Technology, who studies calorie restriction and aging.
But Guarente says Kahn has yet to prove that the same effect is responsible for increased lifespan in calorie-restricted animals. "It might be the same effect or there might be two routes to long life," he points out, "and that would be very interesting./

What does the last sentence in the third paragraph imply（　）

A:People like to lose weight, but they do not like to eat less. B:People want to go to heaven,but they do not want to die. C:Mice will go to heaven if they lose weight. D:Mice enjoy losing weight.

Eat More, Weigh Less, Live Longer
Clever genetic detective work may have found out the reason why a near-starvation diet prolongs the life of many animals.
Ronald Kahn at Harvard Medical School in Boston, US, and his colleagues have been able to extend the lifespan (寿命) of mice by 18 per cent by blocking the rodent’s (啮齿动物) increase of fat in specific cells. This suggests that thinness—and not necessarily diet—promotes long life in "calorie (热量卡) restricted" animals.
"It’s very coal work," says aging researcher Cynthia Kenyon of the University of California, San Francisco. "These mice eat all they want, lose weight and live longer. It’s like heaven."
Calorie restriction .dramatically extends the lifespan of organisms as different as worms and rodents. Whether this works in humans is still unknown, partly because few people are willing to submit to such a strict diet.
But many researchers hope they will be able to trigger the same effect with a drug once they understand how less .food leads to a longer life. One theory is that eating less reduces the increase of harmful things that can damage cells. But kahn’s team wondered whether the animals simply benefit by becoming thin:
To find out, they used biology tricks to disrupt the insulin (胰岛素) receptor (受体) gene in lab mice—but only in their fat cells. "Since insulin is needed to help fat cells store fat, these animals were protected against becoming fat," explains Kahn.
This slight genetic change in a single tissue had dramatic effects. By three months of age, Kahn’s modified mice had up to 70 per cent less body fat than normal control mice, despite the fact that they ate 55 per cent more food per gram of body weight.
In addition, their lifespan increased. The average control mouse lived 753 days, while the thin rodents averaged a lifespan of 887 days. After three years, all the control mice had died, but one-quarter of the modified rodents were still alive.
"That they get these effects by just manipulating the fat cells is controversial," says Leonard Guarente of the Massachusetts Institute of Technology, who studies calorie restriction and aging..
But Guarente says Kahn has yet to prove that the same effect is responsible for increased lifespan in calorie-restricted animals. "It might be the same effect or there might be two routes to long life," he points out, "and that would be very interesting./

What does the last sentence in the third paragraph imply（　）.

A:People like to lose weight, but they do not like to eat less. B:People want to go to heaven, but they do not want to die. C:Mice will go to heaven if they lose weight. D:Mice enjoy losing weight.

Eat More, Weigh Less, Live Longer
Clever genetic detective work may have found out the reason why a near-starvation diet prolongs the life of many animals.
Ronald Kahn at Harvard Medical School in Boston, US, and his colleagues have been able to extend the lifespan (寿命) of mice by 18 per cent by blocking the rodent’s (啮齿动物) increase of fat in specific cells. This suggests that thinness—and not necessarily diet—promotes long life in "calorie (热量卡) restricted" animals.
"It’s very coal work," says aging researcher Cynthia Kenyon of the University of California, San Francisco. "These mice eat all they want, lose weight and live longer. It’s like heaven."
Calorie restriction .dramatically extends the lifespan of organisms as different as worms and rodents. Whether this works in humans is still unknown, partly because few people are willing to submit to such a strict diet.
But many researchers hope they will be able to trigger the same effect with a drug once they understand how less .food leads to a longer life. One theory is that eating less reduces the increase of harmful things that can damage cells. But kahn’s team wondered whether the animals simply benefit by becoming thin:
To find out, they used biology tricks to disrupt the insulin (胰岛素) receptor (受体) gene in lab mice—but only in their fat cells. "Since insulin is needed to help fat cells store fat, these animals were protected against becoming fat," explains Kahn.
This slight genetic change in a single tissue had dramatic effects. By three months of age, Kahn’s modified mice had up to 70 per cent less body fat than normal control mice, despite the fact that they ate 55 per cent more food per gram of body weight.
In addition, their lifespan increased. The average control mouse lived 753 days, while the thin rodents averaged a lifespan of 887 days. After three years, all the control mice had died, but one-quarter of the modified rodents were still alive.
"That they get these effects by just manipulating the fat cells is controversial," says Leonard Guarente of the Massachusetts Institute of Technology, who studies calorie restriction and aging..
But Guarente says Kahn has yet to prove that the same effect is responsible for increased lifespan in calorie-restricted animals. "It might be the same effect or there might be two routes to long life," he points out, "and that would be very interesting./

What does the last sentence in the third paragraph imply（）。

A:People like to lose weight, but they do not like to eat less. B:People want to go to heaven, but they do not want to die. C:Mice will go to heaven if they lose weight. D:Mice enjoy losing weight。

Clever genetic detective work may have found out the reason why a near starvation diet prolongs the life of many animals.
Ronald Kahn at Harvard Medical School in Boston, U.S., and his colleagues have been able to extend the lifespan of mice by 18 per cent by blocking the rodent’s increase of fat in specific cells. This suggests that thinness and hot necessarily diet promotes long life in "calorie restricted" animals.
"It’s very cool work." says aging researcher Cynthia Kenyon of the University of California, San Francisco. "These mice eat all they want, lose weight and live longer. It’s like heaven."
Calorie restriction dramatically extends the lifespan of organisms as different as worms and rodents. Whether this works in humans is still unknown, partly because few people are willing to submit to such a strict diet.
But many researchers hope they will be able to trigger the same effect with a drug once they understand how less food leads to a longer life. One theory is that eating less reduces the increase of harmful things that can damage cells. But Kahn’s team wondered whether the animals simply benefit by becoming thin.
To find out they used biology tricks to disrupt the insulin (胰岛素) receptor (受体) gene in lab mice but only in their fat cells. "Since insulin is needed to help fat cells store fat, these animals were protected against becoming fat," explains Kahn.
This slight genetic change in a single tissue had dramatic effects. By three months of age, Kahn’s modified mice had up to 70 percent less body fat than normal control mice, despite the fact that they ate 55 percent more food per gram of body weight.
In addition, their lifespan increased. The average control mouse lived 753 days, while the thin rodents averaged a lifespan of 887 days. After three years, all the control mice had died, but one-quarter of the modified rodents were still alive.
"That they get these effects by just manipulating the fat cells is controversial," says Leonard Guarente of the Massachusetts Institute of Technology, who studies calorie restriction and aging.
But Guarente says Kahn has yet to prove that the same effect is responsible for increased lifespan in calorie restricted animals. "It might be the same effect or there might be two routes to long life," he points out, "and that would be very interesting./
What does the last sentence in the third paragraph imply

A:People like to lose weight, but they do not like to eat less. B:People want to go to heaven but they do not want to die. C:Mice will go to heaven if they lose weight. D:Mice enjoy losing weight.

{{B}}第三篇{{/B}}

What does the last sentence in the third paragraph imply?

A:People like to lose weight, but they do not like to eat less. B:People want to go to heaven, but they do not want to die. C:Mice will go to heaven if they lose weight. D:Mice enjoy losing weight.

第三篇 Eat More，Weigh Less，Live Longer Clever genetic detective work may have found out the reason why a near。starvation diet prolongs the life of many animals． ‘ Ronald Kahn at Harvard Medical School in Boston，US，and his colleagues have been able to extend the lifespan(寿命)of mice by 18 per cent by blocking the rodent’s(啮齿动物) increase of fat in specific cells．This suggests that thinness--and hot necessarily diet—-promotes long life in “ calerie (热量卡) restricted” animals． “It’s very cool work．”says aging researcher Cynthia Kenyon of the University of California，San Francisco．“These mice eat all they want，lose weight and live longer．It’s like heaven．” Calorie restriction dramatically extends the lifespan of organisms as different as worms and rodents．Whether this works in humans is still unknown，partly because few people are willing to submit to such a strict diet． But many researchers hope they will be able to trigger the same effect with a drug once they understand how less food leads to a longer life．One theory is that eating less reduces the increase of harmful things that can damage cells．But Kahn’s team wondered whether the animals simply benefit by becoming thin． To find out。they used biology tricks to disrupt the insulin(胰岛素)receptor(受体)gene in lab mice—_but only in their fat cells．“Since insulin is needed to help fat cells store fat，these animals were protected against becoming fat，”explains Kahn． This slight genetic change in a single tissue had dramatic effects．By three months of age，Kahn，s modified mice had up to 70 per cent less body fat than normal control mice，despite the fact that they ate 55per cent more food per gram of body weight． In addition，their lifespan increased．The average control mouse lived 753 days，while the thin rodents averaged a lifespan of 887 days．After three years，all the control mice had died，but one--quarter of the modified rodents were still alive． “That they get these effects by just manipulating t11e fat cells is controversial，”says Leonard Guarente of the Massachusetts Institute of Technology, who studies calorie restriction and aging． ． But Guarente says Kahn has yet to prove that the same effect is responsible for increased lifespan in calorie．restricted animals．“It might be the same effect or there might be two routes to long life，”he points out，“and that would be very interesting．” What does the last sentence in the third paragraph imply?

A:People like to lose weight，but they do not like to eat less． B:People want to go to heaven．but they do not want to die． C:Mice will go to heaven if they lose weight． D:Mice enjoy losing weight．